What is Fanconi Syndrome characterized by?

Fanconi Syndrome is characterized by a specific set of clinical features that arise from a defect in proximal tubular function in the kidneys, leading to a loss of various substances in the urine. The correct option highlights the hallmark manifestations of this syndrome, which include anemia, mental retardation, rickets, and aminoaciduria.

Anemia can occur due to the associated renal tubular dysfunction and the resultant decreased erythropoietin production. Mental retardation may be seen in cases where underlying metabolic disturbances or genetic factors are involved. Rickets is linked with the loss of phosphate and impaired vitamin D metabolism, which affects bone mineralization. Aminoaciduria, the excretion of amino acids in urine, is a result of the kidney's inability to properly reabsorb amino acids due to tubular defects.

This combination of symptoms is distinctly identifiable in Fanconi Syndrome and reflects the renal and systemic complications that arise from proximal tubular dysfunction. Other options present unrelated conditions or combinations of symptoms that do not significantly connect to the key features of Fanconi Syndrome, making them less relevant in this context.